Carpel tunnel syndrome
Definition
Collection of symptoms and signs caused by compression of the median nerve in the carpal tunnel
Typical symptoms include numbness and tingling mainly in the first 3 fingers, aching and pain in the anterior wrist and forearm, and clumsiness in the hand
Risk Factors
Age >30
High BMI
Female
Damage to wrist
RA
Diabetes
Square wrist
Dialysis
deposition of amyloid on tendons and other structures within the carpal tunnel
Pregnancy
Congenital carpal tunnel stenosis
Repetitive bending, vibration, twisting of wrist
Smoking
Differential diagnosis
OA
Stroke
C6/C7 radiculopathy
Ulnar neuropathy
Amyotrophic lateral sclerosis
Motor neurone disease
De Quervain's syndrome
Lateral epicondylitis
Rotator cuff tendonitis
Polyneuropathy
Brachial plexopathies
Proximal median neuropathy
MS
Epidemiology
US incidence 3.5 cases per 1000 person-years
Prevalence 3.7%
Appears to be much more rare in developing countries and in other ethnic groups
Rates of CTS have risen considerably over the past several decades, but whether this is due solely to better recognition or a rise in risk factors is debatable
CTS is at least 3 times more common in females
Aetiology
It is rare to find any single definite cause in a patient
Causation is very likely multi-factorial.
Clinical features
Numbness of hand(s)
Median nerve distribution
Weakness in hand / thenar muscle weakness
Median nerve distribution
LOAF
Lumbricals 1+2
Opponens pollicis
Abductor pollicis brevis
Flexor pollicis brevis
Clumsiness
Aching and pain in arm
Worse at night
Intermittent symptoms
Gradual onset
Pathophysiology
The final common pathway is the development of raised pressure within the carpal tunnel and/or the median nerve segment that lies within the tunnel
The increased pressure could trigger off a chain of negative events that ultimately leads to ischaemia and scarring of the nerve
Ischaemia is likely to be the cause of the typical intermittent sensory symptoms of CTS
if it remains intermittent, then no axonopathy occurs
Pressure on the nerve will also lead to demyelination (and ultimately axonal loss), which is the main finding on neurophysiological testing
Demyelination per se causes no symptoms to the patient
This may explain in part the subgroup of individuals who have no symptoms of CTS but who have abnormalities on nerve conduction studies
Investigations
Electromyogram
focal slowing of conduction velocity in the median sensory nerves across the carpal tunnel
prolongation of the median distal motor latency
possible decreased amplitude of median sensory and/or motor nerves
Wrist ultrasound/MRI
Hard to distinguish the median nerve
Management
a) conservative
Wrist splints, especially at night
b) medical
40-mg methylprednisolone acetate (1 mL volume) +/- local anaesthetic is used
The volume should be kept low, as an increase in fluid could exacerbate the already raised pressure in the carpal tunnel
Oral steroids do work, but not as well as intracarpal injection
Potentially a mild diuretic (e.g., hydrochlorothiazide) if wrist oedema present
NSAIDS
c) surgical
Surgical release
Prognosis
Untreated CTS can spontaneously improve in up to one third of individuals, particularly in younger females
Most studies show about a 70% chance of improvement with splints after a period of weeks to months
Most studies show a success rate of between 60% and 70% up to 6 months post-injection with corticosteroids
Surgical success rates are about 80% to 90%