13.05.15 Psychopharmacology
EPSEs
Early and common
Parkinsonism
Akathesia
Dystonia
Late (years-decades), irreversible (in a third), often on withdrawal or reduced dose
Tardive dyskinesia
Antipsychotics
Receptors
Alpha1 NA
Orthostatic hypotension
Sedation
H1
Sedation
mACh
Reduceds EPSEs
Autonomic antimuscarinic effects
Dopamine
D2 receptors (Esp. D4) => Antipsychotic
Anti-EPSEs
Centrally acting antimuscarinics
Benzatropine, orphenadrine, procyclidine
May make TD worse
Clozapine
Hits D4
Is a potent antimuscarinic
But yet causes hypersalivation
? Knocks out swallow reflex (so only apparent at night?)
Atypicals
All have strong anti-5-HT2 activity
Adverse effects:
Metabolic syndrome
Diabetes
Increased QT
Hypotension
Weight gain
Aripiprazole
"Dopamine stabiliser"
Stimulates at low [D]; Inhibits at high
Side-effects
Orthostatic hypotension
Seizures
Somnolence
As effective as risperidone; Not quite as good as olanzapine
Mood disorders
Progression
MAOIs
MAO also in GI tract => "Cheese effect" (tyramines) => Hypertensive crisis
Interaction with indirect sympathomimetics (epedrine etc)
TCAs
Antimuscarinic effects
NA uptake block => Tachycardia
Dangerous + no antidote
Atypicals
SSRIs
Half-lifes 20-30 hours, except fluoxetine (4-16 days) => Long wash-out
Early anxiety, agitation, nervousness
Beware in bipolar! => Manic symptoms
Increase bleeding risk cf NSAIDs, warfarin
P450 inhibition : Fluoxetine > Paroxetine > Citalopram/Sertraline
Discontinuiation syndrome: Dizziness, lethargy, nausea, headache, paraesthesia
SNRIs
Lithium has a very narrow t. window
Mood stabilisers are often antiepileptics (carbamazepine, valproate, lamotrigine)
Notes
Dopamine receptors
D1 = D1, D5
D2 = D2, D3, D4
Serotonin syndrome with SSRI plus MAOI/St John's Wort
All anticonvulsants reduce effectiveness of COCP
Flumazenil reverses benzo effect
Zopiclone is probably also addictive
SSRIs in anxiety disorders