12.12.05 Infection lecture
Infection sites
Osteomyelitis in old men
May well be from disseminated prostatitis
Not Strep/Staph like normal
Strep. milleri
Causes ABSCESSES
Easy-to-miss Staph. aureus
Osteomyelitis + endocarditis
Whereas skin/lines/soft tissue are obvious
Proteus
UTIs
Strep. bovis
Bowel malignancy (tumour allows invasion)
Bacteroides
Peritonitis (from gut flora)
Progression of endocarditis
Non-bacterial thrombotic endocarditis
vWF adheres
Platelets stick
Fibrin mesh
Invasion
=> Vegetation
All starts with TURBULANCE, causing damage
Rheumatic heart disease
Congenital heart disease
Buggered valves
Hyperdynamic circulation?
Management of endocarditis
Cultures
Take 6, from multiple sites
c.f. Low levels, contaminants
Serology
If Pt has already had Abx
To check culture-negative Pt for low-level or weird pathogens
Echo
65-95% sensitivity
Don't rely on echo for diagnosis
Very hard to see prosthetic valve endocarditis on echo
Tends to affect the ring of scar tissue, not the valve itself
Treatment
3-6 weeks of penicillin + gentamicin
Surgery (esp. for prosthetic valve infection)
Abx for dental work??
Viridans Streptococcus
Most abundant in the mouth
S. mutans, is the etiologic agent of dental caries
Others may be involved in other mouth or gingival infections
Most common causes of subacute bacterial endocarditis
Have the unique ability to synthesize dextrans from glucose, which allows them to adhere to fibrin-platelet aggregates at damaged heart valves
Host factors for post-operative infection
Diabetes
Obese
Malnutrition
Smoker
Low albumin
Affects both healing and immune function
Prior irradiation
Prolonged pre-op stay
Steroids
S. aureus carriers
Old and poor ?
Skin infections
Impetigo
Dermis only
=> Topical fusidic acid
Erysipides
Skin plus subcutaneous lymphatics
Raised edge, oedema
Typically GAS
Cellulitis
Skin and subcutaneous tissue
GAS or S. aureus
Be sure to exclude DVT
=> High-dose pen + fluclox. + elevation
Necrotising fasciitis
Organisms
GAS (beta-haemolytic => aggressive spread)
Faecal organisms
Spreads along fascial planes => Systemic sepsis
High mortality
Treatment
Antibiotics including anti-ribosomal agents to block toxin production (not just bacteriostatic agents)
IVIG
Gas gangrene
Typically from anaerobes
e.g. Clostridium, from soil (WW 1)
Now mostly seen in IVDUs
Bacterial taxonomy
Notes
Blood cultures can be falsely +ve in laukaemia due to CO2 production from WBCs
Most common cause of splinter haemorrhages is trauma
e.g. Builders
Osler nodes are sore
Inflammation is a normal part of wound healing!
Don't be scared of a bit of redness
Clenched-fist injury is the same as a bite
Assume they're all infected
Paronychia = Skin infection that occurs around the nails
Skin cannot be sterilised
Puncture wounds look the best but fare the worst