12.11.27 GI bleeds
Locations
Haematemesis
Start: Nose/Mouth
cf Nosebleed
Finish: Ligament of Treitz (D3)
Melena
Start: Nose/Mouth
Finish: Iliocaecal valve
Takes about 14 hours for blood to be broken down within the intestinal lumen
If transit time is less than 14 hours the patient will have hematochezia
If greater than 14 hours the patient will exhibit melena
Ligament of Treitz
Connects the duodenum to the diaphragm
Inserts into the third and fourth portions of the duodenum and frequently into the duodenojejunal (DJ) flexure
When it contracts, the suspensory muscle of the duodenum widens the angle of the duodenojejunal flexure, allowing movement of the intestinal contents
Melena
Sinister symptom
Syncope/pre-syncope
e.g. Went to the toilet, Saw blood, Stood up, Fainted
Relevant drugs
NSAIDS, aspirin, warfarin
Beta blockers - Mask reflex tachycardia
Bad prognostic features
Heart failure
Ischaemic heart disease
Renal failure
Liver failure
Metastatic cancer
Perforated DU
Used to do excision and end-to-end anastomosis (Billroth procedure)
Vulnerable to cancer and re-bleeding
Now overlay layers => Better
Aorto-gastric fistula
From inflamed aorta
e.g. Rejection of AAA stent
May mimic ulcer presentation
But endoscopy negative and omeprazole doesn't help
Then bleed catastrophically
Stigmata of chronic liver disease
Spider naevi
Fill from the centre
Gynaecomastia
Liver can't metabolise oestrogen
Loss of secondary sexual hair
Ascites
Caput medusae
Everted umbilicus
Jaundice
Liver and inflammation
Liver failure => Loss of inflammatory response
Serious risk of asymptomatic spontaneous bacterial peritonitis
Must check with an ascitic tap
Rockall score
A - Age
B - Blood pressure fall (shock)
C - Co-morbidity
D - Diagnosis
E - Evidence of bleeding
Interpretation - Mortality
1 - Nil
2 - Nil
3 - 5%
4 - 5-10%
5 - 5-10%
6 - 5-10%
7+ - 10-35%
Most elderly patients are 25-50% mortality risk from a GI bleed!
Things distinguishing an enlarged kidney from an enlarged spleen on examination
The spleen has no palpable upper border
The space between the spleen and the costal margin (which is present in enlarged kidneys) cannot be felt
The spleen has a notch that may be palpable
Spleen moves inferomedially on inspiration while the kidneys move more inferiorly
Spleen not balottable unless gross ascites present, but kidneys are because of retroperitoneal position
Percussion note is dull over the spleen but usually resonant over kidneys due to overlying bowel
A friction rub may be heard over spleen but not over kidney as it is too posterior
Management of GI bleed
Fluids while you're waiting, but they really need BLOOD
Cryoprecipitate
Expensive but great at replacing clotting factors
Vit K, platelets
Sengstaken-Blakemore tube for varices
Balloon tamponade
Just inflate the gastric (not the oesophageal) balloon, to avoid necrosis
Terlipressin
Splanchnic vasoconstrictor
Reduces HPV blood flow => Less bleeding from varices
Beware gut ischaemia!
SIRS
Systemic inflammatory response syndrome
=> BP rise in response to infection
Can cause varices to pop
=> Manage varices with antibiotics to treat the underlying cause
Lactulose
Used to drop gut pH
Kills bacteria to prevent ammonia production
May not be converted to urea if your liver's broken
=> Encephalopathy
Better than using antibiotics (c.f. C. diff)
Notes
Failed discharge is readmission within 48 hrs
Clot the size of a fist = 500 ml of blood
Courvoisier's law
Because the gall bladder with stones is usually chronically fibrosed and therefore incapable of enlargement
MGMT of Mallory-Weiss tear
Observe o/n then home
So don't misdiagnose as e.g. a bleeding ulcer, which means 5 days in hospital!
Peptic ulcers
Oesophageal
Gastric (=> Cancer)
Duodenal
Be careful coffee-ground vomiting really is!
Not just something brown they've eaten
Melena and haematemesis are both equally life-threatening
Stomach pH is lowest around the pylorus
Shutdown? => Femoral
Don't try to put a central line in the internal jugular as you'll cause a pneumothorax
Raised urea:creatinine after a bleed (high-protein meal)
Lots of antibiotics interfere with warfarin metabolism and raise INR!
The most common GI bleed on the ward is ULCERS
NICE do not support the use of PPIs pre-endoscopy
But it can take days to get one done so give them anyway
High INR alone doesn't cause a bleed
Investigate the CAUSE
TIPS
Transjugular intrahepatic portosystemic shunt
Establishes communication between the inflow portal vein and the outflow hepatic vein
Used to treat portal hypertension
Hb takes a while to fall following a GI bleed
So check again in 4 hours
URINE OUTPUT as a marker of volume status